Julian was born too early, with life-threatening immaturity.
Julian spent a long time in the I.C.U. and in hospitals before the doctors let his mother take the baby home.
Over the next four months, Julian developed without any visible problems. In the fifth month of his life the first diagnoses began to appear, which were added as Julian grew up.
Now Julian is almost 6 years old, and he doesn't respond to his name, doesn't answer questions, doesn't make requests, or often sounds like a question he has been asked. The child does not understand the speech addressed to him, " freezes" on numbers and flipping through pages in books.
Julian does not know the dangers, but he is afraid in perfectly safe situations. Julian does not serve himself, often cries and finds it difficult to calm down, cannot express his pain or desires.
Julian does not know how to communicate with other children, is afraid and sometimes behaves strangely in people's society.
Parents are still looking for the causes of the pathologies and are constantly examining the child.
Recent examinations have shown that Julian has congenital brain hypoxia, which has damaged his neural connections. Julian also has various metabolic disorders and autistic traits.
And because of the shortening of one leg, Julian's gait is very unstable and the baby often falls.
But after every rehabilitation course, Julian is making great progress.Each course is a small victory over a problem, so the treatment cannot be stopped.
The planned rehabilitation course is very important, because Julian has to be prepared for school.
Julian's parents and our charitable foundation have high hopes for supporting people, and together with you we will be able to help Julian on his way to healing.
Julian Sentsov, born on 22.07.2014, Krasnodar city.
Diagnosis: Organic brain damage (hypoxic hemorrhagic infectious genesis). CEREBRAL CEREBRAL PALSY. Lower spastic paraparese. Eye movement disorders, MSD and autism-like elements in behavior.
HCG, subcompensated. Carrier of type 6 GHG. Functional dyspepsia. Malabsorption syndrome. Gallbladder shape anomaly.
